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Foods that affect clonazepam



Question regarding Xanax and food benzodiazepines

9.13.2018 | Aidan Jerome
Foods that affect clonazepam
Question regarding Xanax and food benzodiazepines

This is a really specific question, nobody is going to be able to l you "wait X time to eat and it will have 0 effects on the high" that's not really how it works, we don't know your (drug/eating/workout) habits, body type/chemistry and so on to be able to give an answer. Eat and see how you feel. If your high.

Thanks a lot dude/dudette. I actually like this answer.

i dunno though, could be totally wrong. for some reason, probably just me personally, i find milkshakes or anything that makes you feel 'full' enhances the high. and, usually i pig out. ive eaten bars for years.

I can agree with this point.

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Clonazepam-induced hyperphagia in nondeprived rats tests of

8.12.2018 | Kayla Bishop
Foods that affect clonazepam

Nondeprived male rats were familiarised with daily 60 min access to a highly palatable diet, consisting of powdered rat diet, sweetened condensed milk and water. Clonazepam (0.625-5.0 mg/kg, IP) produced a substantial increase in food consumption within the first 30 min of access. The increase was similar across all.

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The pyrazoloquinoline, CGS 9896, binds with high affinity to benzodiazepine sites and has recently been described as a nonsedating anxiolytic. Instead, food consumption was significantly depressed following the administration of Ro5-4864 at 20 and 40 mg/kg, IP. Comparison with the results for Ro5-4864 rules out an interpretation for the anorexia in terms of anxiogenic effects.(ABSTRACT TRUNCATED AT 250 WORDS). The atypical 1,4-benzodiazepine, Ro5-3663, a GABA antagonist which may act at the picrotoxinin site, produced a dose-related reduction in food consumption. A comparison of the clonazepam and Ro5-4864 data suggests that benzodiazepine-induced hyperphagia is mediated by central-type benzodiazepine binding sites. CGS 9896 (2.5-20.0 mg/kg, administered either IP or PO) did not affect consumption of the highly palatable diet. Clonazepam (0.625-5.0 mg/kg, IP) produced a substantial increase in food consumption within the first 30 min of access. Treatments with the peripheral-type benzodiazepine agonist, Ro5-4864, did not induce a hyperphagic response. The increase was similar across all dose conditions, suggesting that a maximal effect may have been achieved with a dose as small as 0.625 mg/kg. Nondeprived male rats were familiarised with daily 60 min access to a highly palatable diet, consisting of powdered rat diet, sweetened condensed milk and water. The hyperphagia induced by clonazepam was reversed by the benzodiazepine receptor antagonist, Ro15-1788 (5.0-20.0 mg/kg), indicating that the effect was benzodiazepine receptor-mediated. In consequence, anxiolytic and hyperphagic effects of drug actions at benzodiazepine receptors may be dissociated in the case of this compound.

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